10/24/2020

Posterior Tibial Tendon Dysfunction (PTTD)

Posterior Tibial Tendon Dysfunction (PTTD)

Posterior tibial tendon dysfunction (PTTD), also known as posterior tibial tendonitis, is one of the leading causes of acquired flatfoot in adults. The onset of PTTD may be slow and progressive or sudden. An abrupt starting point is usually linked to some form of trauma, whether it be simple (stepping down off a curb or ladder) or severe (falling from a height or car accident). PTTD is hardly ever seen in children and increases in frequency with age.

The Characteristic Finding of PTTD Include;

Loss of medial arch height.

Edema (Swelling) of the Medial Ankle

Loss of the ability to resist force to be able to abduct or push the foot out from the midline of the body.

Pain on the Medial Ankle With Weight Bearing

Inability to boost up on the foot without pain.

Too Many Toes Sign

Lateral subtalar joint (outside of the ankle) pain.

Common test to evaluate PTTD could be the 'too many feet sign'. The too many toes sign' is a test used to measure abduction deviation away from the midline of the body) of the forefoot. With damage to the rear tibial tendon, the forefoot will abduct or move out in relationship to the rest of the foot. In cases of PTTD, if the foot is viewed from guiding, the toes appear as 'too many' on the outside of the foot due to abduction of the forefoot.

Advanced cases of PTTD, in addition to the pain of the tendon itself, pain will also be noted at the sinus tarsi. The nose tarsi refers to a small canal or divot on the outside of the ankle that can actually be felt. This tunnel is the entry to the subtalar joint. The subtalar joint is the joint that controls the side to side motion of the foot, motion that would occur with uneven surfaces or sloped hills. As PTTD progresses and the ability of the rear tibial tendon to support the arch becomes reduced, the arch will collapse overloading the subtalar shared. As a result, there is increased pressure placed on the joint floors of the lateral aspect of the subtalar joint, resulting in soreness.

There have been many proposed explanations for PTTD over the years because this condition was first described by Kulkowski inThe most modern day explanation refers to an area of hypovascularity (limited blood flow) in the tendon just below the ankle. Tendon comes nearly all of its' nutritional support from synovial fluid produced by the particular outer lining of the tendon. Really small blood vessels also permeate the tendons sheath to arrive at tendons. This makes all tendon notoriously slow to be able to heal. In the case of the posterior tibial tendon, this problem is exacerbated by a distinct section of weak blood flow hypovascularity). This area is located in the posterior tibial tendon just below or distal to the inside ankle bone (medial malleolus).

Tendon is also many prone to fatigue and failure at an area in which the tendons changes direction. As the posterior tibial tendon descends the leg and comes to the inside of the ankle, the tendon follows a well defined groove in the back of the tibia (bone of the inside of the ankle). The tendon then takes a dramatic turn towards the arch of the foot. If the muscle is put into a situation where significant load is applied to the foot, the tendon responds by pulling up as the load of the body (in addition to be able to gravity) pushes down. At the location where the tendon modifications course, the tibia acts as a wedge and could utilize enough force to actually damage or break the tendon.

Equinus is Also a Contributing Factor to PTTD

Equinus is the term used to describe the ability or lack of ability to dorsiflex the feet on the ankle (move the toes toward you).Equinus is usually as a result of tightness in the leg muscle mass, also known as the gastroc-soleal complex (a combination of the gastrocnemius and soleus muscles). Equinus may also be due to a bony block in the front of the ankle. The presence of equinus makes the rear tibial tendon to accept additional insert during gait.

Additional contributing factor to the onset of PTTD may include hypertension, diabetes, peripheral neuropathy, smoking or arthritis.

  • The progression of PTTD may well lead to tendonitis, partial tears of the tendon or perhaps complete tendons rupture.
  • Several classifications have been developed to describe PTTD.
  • The classification as described by Johnson and Strom is most commonly used today.
  • Stage I Tendon status Attenuated (lengthened) with tendonitis but no rupture Clinical findings Palpable pain in the medial arch.
  • Foot is supple, versatile with way too many feet indication X-ray/MRIMild to moderate tenosynovitis on MRI, no X-ray changes
  • Stage II Tendon status Attenuated with possible partial or complete rupture Clinical findings Pain in arch.
  • Not able to raise on toes.
  • A lot of toes indication present X-ray/MRI MRI notes tear in tendon.
  • X-ray noting abduction of forefoot, collapse of talo-navicular joint

Stage III Tendon status Severe degeneration with likely ruptureClinical findings Rigid flatfoot together with inability to raise up on toes X-ray/MRI MRI shows tear in tendon. X-ray noting abduction of forefoot, collapse of talo-navicular joint.

  • Treatment for PTTD is dependant upon the clinical stage and the health status of the patient.
  • It is important to recognize thatPTTD is a mechanical problem that needs a mechanical solution.
  • This means that treating PTTD with treatment on it's own is fraught with failure.
  • Timely introduction of some form of mechanised support is imperative.

Surgical procedures that focus on primary repair of the posterior tibial tendon have been very unsuccessful. This is due to the fact that tendon heals slowly following injuries and cannot be relied upon as a sole solution for PTTD cases. Operative success is usually attained simply by stabilization from the rearfoot subtalar joint) which significantly reduces the work done by the posterior tibial muscle.

Stage I Might Respond to Relaxation, Such as a Walking Throw

Pain and inflammation could be controlled with anti-inflammatory medications. It is important to be sure that Stage I patients realize that the use of shoes with additional arch support as well as heel elevation, for the rest of their lives, is actually imperative. Arch support, whether built into the shoe or added as an orthotic, helps support the posterior tibial muscle and decrease its' work. Elevation of the heel, reduces equinus, one of the most significant contributing factors to PTTD. If Stage I patients come back to low heels without arch support, PTTD will recur.

Stage II patients, or Stage I patients that do not respond to rest and help, require surgical correction to be able to strengthen the subtalar joint prior to further damage to the posterior tibial tendon. Subtalar arthroeresis is a procedure used to stabilize the subtalar joint. Arthroeresis is a term that means the motion of the joint is blocked without fusion. Subtalar arthroeresis can only be used in cases of Stage I or II where mild to moderate deformation of the arch has occurred and MRI findings show the tendons to be only partially ruptured. Subtalar arthroeresis is typically performed in conjunction with anAchilles tendon lengthening procedure to fix equinus. These treatments require casting for a period of weeks following the procedure.

Stage III patients require stabilization of the rearfoot with procedures that fuse the primary joints of the arch and foot. These kinds of procedures are salvage procedures and require prolonged casting and disability following surgery. A common procedure forStage III is called triple arthrodesis which is a technique used to fuse the particular subtalar shared, the talo-navicular joint and the calcaneal cuboid joint.

PTTD is a condition that increases in frequency with age and the prevalence of poor health indicators such as diabetes and obesity. As a result, many patients with PTTD are bad surgical applicants for correction of PTTD. Prosthetics such as an ankle foot orthotic (AFO), Arizona Brace or other bracing may be very helpful to control the symptoms of PTTD. Anatomy:

The posterior tibial tendon is the extension of the posterior tibial muscle that lies deep to the leg. The origin of the posterior tibial muscle is the posterior aspect of both the tibia and fibula and the interosseus membrane. The insertion of the posterior tibial muscle is the medial navicular in which the tendon divides into nine different insertion site on the bottom of the foot.

Biomechanics:

The function of the posterior tibial tendon is always to plantarflex the base on the toe off phase of the gait cycle and to stabilize the medial arch.

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Symptoms:

The symptoms of phase I PTTD include a dull ache of the medial arch. The pain become worse with activity, better on days with limited time on the feet. Extensive activity may result in a partial rupture of the tendon, relocating to stage II.

Gout

  • Stage II signs and symptoms are seen with more regularity.
  • Pain is present at the onset of standing and walking.
  • Some limitation of a chance to raise up on the feet will be present.
  • Stage III symptoms are severe with an inability to complete most normal daily activities such as washing or going to the store.
  • Collapse of the medial arch will be obvious.
  • Abduction of the forefoot will show 'too many toes sign'.

Differential Diagnosis:

Conditions that may resemble PTTD include tarsal tunnel syndrome, tibial stress fractures, posterior tibial muscle rupture, flexor hallucis longus tendonitis, gout, arthritis of the subtalar joint or a fracture of the posterior process of the actual talus.

Additional References Include;

Cantanzariti, A.R., Lee, M.S., Mendicino, R.W. PosteriorCalcaneal Displacement Osteotomy for Adult Acquired Flatfoot. J.of Foot and Ankle Surgery. 39-1: 2-14, 2000

  • Myerson, M.S., Corrigan, J.
  • Treatment of posterior tibial tendons dysfunction with flexor digitorum longus muscle transfer and calcaneal osteotomy.
  • Orthopedics 19:383-388, 1996

Myerson, M.S. Adult acquired flatfoot deformity. J. Bone andJoint Surgery. 78-A;780, 1996

Johnson, K.A., Tibialis posterior tendons rupture. Clin. Orthop. 177:140-147, 1983

About the Particular Author:Jeffrey a

Oster, DPM, C.Ped is a board certified foot and ankle surgeon. Dr. Oster can also be board certified in pedorthics. Doctor. Oster is medical director of Myfootshop.com and is in active practice in Granville, Ohio.